Syracuse University at SfN 2026

Nov 14–18, 2026 Washington, DC ~21k attendees Website

Syracuse University brings 10 presentations to SfN 2026 in Washington. Its SfN 2026 presentations centre on tau-related neurodegeneration, restoring lysosome function, and neuroinflammation in multiple sclerosis and other disease models. One thread explores acidic nanoparticles that restore lysosomal acidification and reduce pathology linked to tau, alpha-synuclein, and amyloid-beta.

Organization	SfN 2026 Attendance
Syracuse University Syracuse, NY
syr.edu Universities and research institutes Private research university in Syracuse, New York, with biology and interdisciplinary neuroscience programs.	Talk Sat Nov 14 1:30–1:45pm Part of Session NANO01: Alzheimer's Disease Therapeutic Strategies: Amyloid, Tau, and Metabolism Inhibition of tau aggregation by a BBB-penetrating peptide to mitigate neurotoxicity in cellular and mouse model of tauopathies WCC Rm 145 Talk Sat Nov 14 2:15–2:30pm Part of Session NANO01: Alzheimer's Disease Therapeutic Strategies: Amyloid, Tau, and Metabolism Lysosome-acidifying nanoparticles with BBB-penetrating capability mitigate tau-driven neurotoxicity and neurodegenerative pathology WCC Rm 145 Poster Sun Nov 15 8am–noon Part of Session PSTR054: Translational Studies and Therapeutic Strategies in Multiple Sclerosis Dissecting Inflammatory and Metabolic Drivers of Neurodegeneration in Secondary Progressive Multiple Sclerosis WCC Halls A-C · Board Y9 Poster Mon Nov 16 8am–noon Part of Session PSTR127: Alzheimer's Disease Therapeutic Strategies: Tau Acidic nanoparticles as a lysosome restoration strategy to attenuate tau-mediated neurotoxicity in cellular and mouse models of tauopathy WCC Halls A-C · Board T27 Poster Mon Nov 16 1–5pm Part of Session PSTR172: Tauopathies, Synucleinopathies, and Other Related Diseases: Animal Models TNFR1 allosteric inhibition by peptide FKC restores neuronal viability and reduces tau pathology in tauopathy models WCC Halls A-C · Board T14 Poster Mon Nov 16 1–5pm Part of Session PSTR204: Multi-Omics Comparative Proteomics Reveals Cell Type-Specific Heterogeneity of Human Neural Cell-Derived Extracellular Vesicles WCC Halls A-C · Board LL18 Poster Tue Nov 17 8am–noon Part of Session PSTR223: Neuroinflammation: Novel Models, Methods, and Therapeutics A TNFR1-selective peptide allosteric inhibitor with blood-brain barrier-penetrating capability attenuates neuroinflammation and neurodegenerative pathology in a chronic inflammation mouse model WCC Halls A-C · Board X7 Poster Wed Nov 18 8am–noon Part of Session PSTR308: Tauopathies, Synucleinopathies, and Other Related Diseases: Cellular and Molecular Mechanisms Structural remodeling of tau aggregation by small molecules to attenuate tau-induced neurotoxicity WCC Halls A-C · Board X13 Poster Wed Nov 18 8am–noon Part of Session PSTR308: Tauopathies, Synucleinopathies, and Other Related Diseases: Cellular and Molecular Mechanisms BBB-penetrating acidic nanoparticles restore lysosomal acidification and reduce α-Synuclein pathology in familial Parkinson’s disease WCC Halls A-C · Board X24 Talk Wed Nov 18 8:45–9am Part of Session NANO39: Alzheimer's Disease: Abeta, APOE, and Tau Mechanisms Lysosome-acidifying nanoparticles restore lysosomal acidification and mitigate Aβ42 oligomer-induced neurotoxicity in cellular and mouse models of Alzheimer’s disease WCC Rm 207A

Organization

SfN 2026 Attendance

Syracuse University

Syracuse, NY

syr.edu

Universities and research institutes

Private research university in Syracuse, New York, with biology and interdisciplinary neuroscience programs.

Talk

Sat Nov 14
1:30–1:45pm

Part of Session NANO01: Alzheimer's Disease Therapeutic Strategies: Amyloid, Tau, and Metabolism

Inhibition of tau aggregation by a BBB-penetrating peptide to mitigate neurotoxicity in cellular and mouse model of tauopathies

WCC Rm 145

Talk

Sat Nov 14
2:15–2:30pm

Part of Session NANO01: Alzheimer's Disease Therapeutic Strategies: Amyloid, Tau, and Metabolism

Lysosome-acidifying nanoparticles with BBB-penetrating capability mitigate tau-driven neurotoxicity and neurodegenerative pathology

WCC Rm 145

Poster

Sun Nov 15
8am–noon

Part of Session PSTR054: Translational Studies and Therapeutic Strategies in Multiple Sclerosis

Dissecting Inflammatory and Metabolic Drivers of Neurodegeneration in Secondary Progressive Multiple Sclerosis

WCC Halls A-C · Board Y9

Poster

Mon Nov 16
8am–noon

Part of Session PSTR127: Alzheimer's Disease Therapeutic Strategies: Tau

Acidic nanoparticles as a lysosome restoration strategy to attenuate tau-mediated neurotoxicity in cellular and mouse models of tauopathy

WCC Halls A-C · Board T27

Poster

Mon Nov 16
1–5pm

Part of Session PSTR172: Tauopathies, Synucleinopathies, and Other Related Diseases: Animal Models

TNFR1 allosteric inhibition by peptide FKC restores neuronal viability and reduces tau pathology in tauopathy models

WCC Halls A-C · Board T14

Poster

Mon Nov 16
1–5pm

Part of Session PSTR204: Multi-Omics

Comparative Proteomics Reveals Cell Type-Specific Heterogeneity of Human Neural Cell-Derived Extracellular Vesicles

WCC Halls A-C · Board LL18

Poster

Tue Nov 17
8am–noon

Part of Session PSTR223: Neuroinflammation: Novel Models, Methods, and Therapeutics

A TNFR1-selective peptide allosteric inhibitor with blood-brain barrier-penetrating capability attenuates neuroinflammation and neurodegenerative pathology in a chronic inflammation mouse model

WCC Halls A-C · Board X7

Poster

Wed Nov 18
8am–noon

Part of Session PSTR308: Tauopathies, Synucleinopathies, and Other Related Diseases: Cellular and Molecular Mechanisms

Structural remodeling of tau aggregation by small molecules to attenuate tau-induced neurotoxicity

WCC Halls A-C · Board X13

Poster

Wed Nov 18
8am–noon

Part of Session PSTR308: Tauopathies, Synucleinopathies, and Other Related Diseases: Cellular and Molecular Mechanisms

BBB-penetrating acidic nanoparticles restore lysosomal acidification and reduce α-Synuclein pathology in familial Parkinson’s disease

WCC Halls A-C · Board X24

Talk

Wed Nov 18
8:45–9am

Part of Session NANO39: Alzheimer's Disease: Abeta, APOE, and Tau Mechanisms

Lysosome-acidifying nanoparticles restore lysosomal acidification and mitigate Aβ42 oligomer-induced neurotoxicity in cellular and mouse models of Alzheimer’s disease

WCC Rm 207A

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Not affiliated with the Society for Neuroscience. Organization details are derived from SfN’s published exhibitor list, its Neuroscience Meeting Planner and each organization’s own website. Submit a correction